Pharaoh 10 ml

Pharaoh, 10 ml 

A unique two-component insecticide used to protect vegetable and fruit and berry crops from a wide range of insects, soil pests, and mites. The product penetrates the treated plant and spreads throughout all its parts, allowing it to kill pests not only on the leaf but also underneath it, as well as in hard-to-reach areas.

Properties and benefits:

• Eliminates a wide range of chewing, mining, and sucking pests.
• Effectively suppresses pest resistance that develops during the intensive use of insecticides from a single chemical group.
• Possesses powerful fumigant activity, allowing it to eliminate pests living in hidden or concealed locations, thereby surpassing many other insecticides in terms of effectiveness.
• Provides extremely rapid action, which in turn reduces dependence on weather conditions.
• Ensures rapid pest elimination (a significant portion of the pest population dies within 24 hours of treatment).
• The fumigant effect is an ideal mechanism for controlling locusts, moths, and other insects that experience population surges.
• Possesses an acaricidal effect.
• Two hours after spraying, precipitation or heavy dew does not reduce the product’s effectiveness.
• Active across a wide temperature range.
• Does not exhibit phytotoxicity to crops when used according to the application guidelines.
• Residual efficacy period: 10 to 14 days.
• Remains active at low temperatures.
• Does not cause insects to develop resistance to the product.

Mode of action 

Once inside the body, chlorpyrifos phosphorylates certain substrates. One such substrate is a protein enzyme found in nervous tissue—acetylcholinesterase (AChE)—which plays a crucial role in the transmission of nerve impulses. AChE belongs to the group of carboxylic acid ester hydrolases. It is localized primarily in receptors on the postsynaptic membrane of the synapse and partly in the membrane of the axon—the projection of the neuron. Chlorpyrifos, by interacting with esterases, suppresses their activity through competitive inhibition. The enzyme acetylcholinesterase hydrolyzes acetylcholine. When the enzyme is blocked by the pesticide, acetylcholine accumulates in the synaptic cleft, leading to disruption of nerve impulse transmission. This results in convulsive muscle activity (tremor), which progresses to paralysis. Chlorpyrifos, entering the insect’s body through the respiratory tract, stomach, or cuticle, reduces the activity of acetylcholinesterase by mimicking acetylcholine. By interacting with AChE, it blocks its action for an extended period. Lambda-cyhalothrin, like other pyrethroids, acts on calcium metabolism in synaptic sodium-potassium channels, disrupting nervous system function. This leads to a significant excess release of acetylcholine during the transmission of nerve impulses. Poisoning manifests as damage to motor centers and severe agitation

Application method and application rate